GC−MS analysis of 786-O and isogenic SETD2-deficient 38E/38F

GC−MS analysis of 786-O and isogenic SETD2-deficient 38E/38F cells. (A)

Fatty Acid Methyl ester GC-MS Mixture

177 Lu-DOTATATE selectively killed hSSTr2 + ESCs in vitro. (A) hSSTr2 +

summarises engraftment, vector copy number (VCN), phenotypical analyses

Hepatic lipid profile in the LDLR knockout rats. Lipids were extracted

RNA-seq analysis of 786-O and isogenic SETD2-deficient 38E and 38F

SETD2 loss-of-function promotes renal cancer branched evolution through replication stress and impaired DNA repair

ZFN activity detection in goat fibroblast cells. a Schematic overview

PDF) Loss of SETD2 Induces a Metabolic Switch in Renal Cell Carcinoma Cell Lines toward Enhanced Oxidative Phosphorylation

PDF) Loss of SETD2 Induces a Metabolic Switch in Renal Cell Carcinoma Cell Lines toward Enhanced Oxidative Phosphorylation

PDF) Loss of SETD2 Induces a Metabolic Switch in Renal Cell Carcinoma Cell Lines toward Enhanced Oxidative Phosphorylation

Sensitive LC-MS/MS Bioanalytical Quantitation of Antisense Oligonucleotides

Determination of enantiomeric and stable isotope ratio fingerprints of active secondary metabolites in neroli (Citrus aurantium L.) essential oils for authentication by multidimensional gas chromatography and GC-C/P-IRMS - ScienceDirect